Are parasites and pathogenic bacteria more common and harmful than we think? (Part 2)

This is a summary of my scientific paper presented at the International Conference and Scientific Debating Club in Prague in April 2014. It elaborates as to why we have to make every effort to avoid parasites and pathogenic bacteria and if they are present  how to eradicate them.

 In Part 1 Why Parasites are harmful to you long term health I have discussed the most common pathogens in the gut  and other affected body organs. One of them is Enterococcus faecum. Depending on where it is located it can be commensal or pathogenic.

Enterorococus faecum commonly present in the gut traditionally has been considered a low grade pathogen, which is part of normal human faeces.17 Sites less colonised are the oral cavity, genitor-urinary, skin and intraabdominal. The ratio of infection due to Enterorococus faecum has increased from historical 10:1 to 1.9:1 in 1999. Sood et al, states that antibiotic fed poultry, cattle, and pigs has caused human antibiotic resistance. Enterorococus faecum is resistant to penicillin and other antibiotics. It has also acquired resistance via mutation of DNA. There are limited therapeutic options in medicine for treatment of serious infections of Enterorococus faecum therefore there is still a challenge for the medical establishment.

The Metatron Hunter has detected high concentration of Enterorococus faecum in patients in the large intestine but also in small intestines, pancreo-duodenum and stomach, and low in blood and oral cavity which is consistent with Sood’s research. Such patients are working either in hospitals, with large groups of people (childcare) or by eating meat from cattle feed with antibiotics. It was evident that the stomach of such a patient was inflamed, thus compromising digestion.

Escherichia coli was detected by the Metatron in high amounts in the entire digestive tract, including stomach, liver, gallbladder, and pancreas and pancroduadenal area. E coli are very diverse strains and some are important members of normal gut flora in humans.5 There are only six distinct groups of E coli (Metatron does not differentiate that) which are pathogens that cause diarrhoea. E coli genome duplicates every 20-30 minutes, incubation is 1-7 days, duration of watery stool is 2-10 days.5 None of the patients presented with those symptoms so no pathogenic species of E coli were suspected.

Clostridium species infection in humans is also often misdiagnosed. Accurate diagnosis requires an antigen test and ova cyst examination of stool. Clostridium perfringens emits “epsilon” toxins which may trigger multiple sclerosis.

Helicobacter pylori appears to be under diagnosed as well. Stomach colonisation of Helicobacter is asymptomatic in most people.¹ Yet, Helicobacter causes gastritis and stomach cancer.¹ The presence of Helicobacter was detected by Metatron not only in both asymptomatic and symptomatic patients but also in 30 % of all Helicobacter- asymptomatic and undiagnosed patients undergoing Metatron health check in our clinic.

Helicobacter pylori in the stomach

Method of research

Six patients (Table 1) with chronic conditions were selected and body scanning and treatments with Metatron Hunter were undertaken every four weeks. Each patient had a different medical condition and prior to the Metatron body scanning and treatment it was not evident what the trigger/s was in their condition.

The composition of gut flora found by the Metatron was analysed and a direct correlation was found between the intake of fibre, animal products and bacteria. Bactericides fragilis was found most of the time in patients who had an animal protein diet.7 As there was only one vegetarian in the group. no conclusion could be made whether Prevotella genus were prevalent.

The symptoms of patients were matched against all findings of Metatron, orthodox tests and clinical observations.

Patients were scanned with Metatron at each consultation and litotherapy, metatherapy, and homeopathic remedy was made by the Metatron. Also herbal medicine, nutritional supplements were dispensed and lifestyle changes were made including reduced consumption of meat and increase intake of soluble and insoluble fibre.

Patients were reminded of the acquired faecal–oral route of contamination of food, fluid, hands; mouth and herbal hand sanitiser, and mouth wash were dispensed and were advised of better choices of food outlets.

Obtained results

Due to low hydrochloric acid or its suppression by proton pump inhibitors, poor diet and lifestyle or occupational exposure, the Metatron has detected Enterococcum faecum in the small intestines pancreo-duodenal area and in the stomach, oral cavity and blood.

The composition of the gut microbiota has improved and changed after NLS treatment in conjunction with the natural treatment.

The more compromised digestive system (the liver, pancreas, pancreo- duodenum, stomach, intestines) the greater was pathogen infestation.

The higher the infestation with Ascaris the greater the food sensitivities. There was no evidence in the studied group that lack of parasitic infestation, “hygiene hypothesis,” leads to autoimmunity and allergies but rather the opposite.

Microbiota composition of a patient is not stable and depends on the diet and other factors as discussed. Therapeutic manipulation of intestinal microbiota16 in the treatment of chronic medical conditions with Lactobacillus, Bifid bacterium spp, Saccharomyces bulgaris or other beneficial bacteria can also be monitored with the Metatron.

Helicobacter pylori was detected in 5 out of 6 patients who had severe to medium HP infections or no past history of Helicobacter. It raises concerns of its consequences such as stomach ulcer or stomach cancer being undetected.

Patients clear of Helicobacter or with no history of Helicobacter had acute or chronic stomach lining with and without the presence of Enteerococcum faecum, which raises a question of what actually causes such inflammation.

Discussion of the results

None of the health conditions, whether acute or chronic, can be fully understood or resolved without an understanding of microbiota and careful balancing pathogenic and non-pathogenic organisms.

From the naturopathic point of view every disease originates from digestive problems and status of microbiota. This view is also well supported in recent scientific medical literature. This was demonstrated not only in 6 studied cases, but all patients in our clinic who undertook NLS body scanning so far.

Early detection of intestinal dysbiosis by the Metatron can prevent and minimise the risk factors of serious Enteerococcum faecum infections or SIBO so that conventional medicine or natural medicine intervention can be undertaken.

Helicobacter pylori infection often goes undiagnosed and the Metatron has proven to be effective in identifying and eradicating without the use of antibiotics.

The Metatron body scanning for pathogens may be the first line of detection and treatment before /if pathology tests or other investigations are undertaken.

There is great scope of prevention of chronic and degenerative conditions such as Ulcerative colitis, Crowns’ disease, diabetes, fatty liver, colon and rectal cancers or multiple sclerosis (MS) and an early detection of intestinal dysbiosis and Metatron can offer such information.


NLS technology has not only opened up a new insight into the human body, but also revealed its correlation between the status of microbiota and chronic conditions. A practioner has instant access to the inside a patient’s body organ cell, tissue, and microbiota at their fingertips. Metatron can be the first line of investigation before time consuming and costly stool analyses or colonoscopies. Those tools can be applied at a later stage if required.

Colon or bowel cancers are on the increase worldwide and Metatron may be the invaluable tool in early detection of microbiota and gut changes. Through application of NLS and mapping a patient’s dynamic microbiota chronic, degenerative or autoimmune conditions like IBD, Crohn’s, and Ulcerative colitis – SIBO or MS can be prevented. The dynamic microbiota of the patient can now be monitored with the Metatron in a non-invasive and fast method. Further investigation of human microbiota with the Metatron may reveal more about the link between gene expression and exposure to pathogens.

There are challenges in addressing unresolved inflammation among orthodox and natural medicine. Metatron may assist in that challenge as well by identifying and monitoring microbiota and the body organs, tissue, cell or perhaps even DNA response.

Table 1

PatientReason for consultation


Other symptoms arising at consultationMetatron intestinal scanningOrthodox testsOrthodox


1.CF, Female

65 y old


IBSArthritis, asthma, allergies, sinusitis,

Inability to put on weight, restless leg syndrome

Helicobacter pylori( HP)– severe

Dientaomeba fragilis-large intestine.

Klebsiella pneumonia, Enterococcum faecum in stomach

Bacillus cereus

E. coli

low eosinophils on blood countSomac




( reduce BP)

2.RJ, Female

44 y old,

IBS, Fatigue

Abdominal pain, diarrhoea

Hot flushes, refluxHP, Erosive gastritis,

Ascaris lumbricoides, Enterrobius vermicularis, Chlamydia, Candida. Bacillus cereus,

E. coli

Upper GI endoscopy-

Epigastric and right upper quadrant, sub umbilical and right quadrant pain

Nexium,10mg ( 2 weeks) Vancomycin 250,(severe infections ) Rifaximin 500,( killing bacteria in gut )  Metronidazole 200(infection by bacteria  and other microorganisms)
3.AP, Male, 28

y old

Stomach ulcer, brain fog, muscle pain, severe food intolerance and allergies

Positive HP

Adrenal fatigue, IBS, heartburn, nauseaSevere infestation with round worm & pin worm, medium bacillus cereus, HP, adenovirus, E.coliDry blood spot for allergens, methane breast test. Stool culture( fructose malabsorption)

stool test for HP positive , but urea  breath negative

Prescribed  Nexium  for 7days but not taken
4.CB, Female 66 y old, vegetarian, childcare workerFatigue

Ulcerative colitis, bloody diarrhoea,


Rapid weight loss of 14kgBacterium proteus

Candida albicans, Streptococcus

haemolyticus A. Streptococcus agalactia B


Serum iron below min low trasferrin saturation

Faecal culture- Blastocystis homins

 Endolimax nana isolated

Colonoscopy –pin worm

 HP in the past


( UC treatment and relapse) Salazopirin (immune suppressant and anti-inflammatory)

5.FT, Female

59 Y old

Chronic diarrhoea, rapid weight lossDiverticulitis


Bactericides fragilis in gut and liver,

Enteerococcum faecum in pancreo-duodenal area, stomach,


Stool analyses for Dientaomeba fragilis, Guardia, cryptosporidium, Salmonella, Vibriospp, Aeromonas, hydrophilic, Shigella Caphylobacter , Plesiomonas shigelloides negativeNone
6.NP Male 40 y oldfatigueHair loss. Stomach ulcerAtrophic gastritis,

Salmonella typhimurium

 Ascaris, Bacterium proteus, Bacterium lactis aerogenes, Bacillus cereus, Rotavirus, Dientaomeba fragilis

HP negativeNone


Metatron image of the stomach of the Patient FT 4

48 % improvement of the gut function


Undiagnosed Helicobacter pylori, 3 week apart with Metatron treatment, 86 % improvement

If you would like to know more about how the DH-Natural Medicine Clinic can help you,  please call us now on (02) 9541 2428 to book your free 15 minute phone consultation.

Danuta Hulajko is a holistic practitioner, international speaker and the founder & practitioner at the DH Natural Medicine Clinic and in Sydney.

Danuta specialises in Allergies, Anti-Aging, Auto-Immune Conditions, Cardiovascular Conditions, Female Reproductive, Menopause, Mould Toxicity, Skin Conditions, Stress and Insomnia and Thyroid Dysfunction.

For more information please go to our website. You can also follow Danuta Hulajko’s work, events, seminars, expos, latest health research, her health tips and advice on FacebookLinkedIn and Instagram.



  1. Cerf-Bensussan N. et al, The immune system and the gut microbiota: friends or foe? Nature Reviews Immunology 2010, Oct:10 (10):735-44
  2. Compare D, et al, Effects of long-term PPI treatment on producing bowel symptoms and SIBO, European Journal of Clinical Investigation, 2011,April Vol 41(4) :380-6
  3. G, A new vision of immunity; homeostasis of the superorganism, Nature, September 2010, Vol 3, Number 5
  4. European Association for the Study of the Liver. “Gut microbiota transplantation may prevent development of diabetes and fatty liver disease.” ScienceDaily,19 April 2012. <>
  5. R. V. ,Dockrel lH. M.,Zuckerman M., Roit I. M., Chiodini P. L, Mim’s Medical Microbiology, Fifth Edition, 2013.Elsevier
  6. Harnet W, Harnett MM.Therapeutic immunomodulators from parasites. Expert Rev Mol Med, June 2008; Vol 10; e18
  7. Jefferey I,B., O”Toole P.W., Diet –Microbiota I interactions and their implications for healthy living. Nutrients 2013, 5,234-252
  8. LindenJR,at al. Epsilon toxin: An Environmental Cause of Multiple Sclerosis” ASM Biodefense and Emerging Disease Research Meeting Clostridium perfringens 2014
  9. MayerLMucosal immunity, Paediatrics’. 2003, June ;111(6 Pt3):1595-600
  10. G.L, Elson, Ch .O., Weaver H, Weaver C., Reciprocal integrations of the intestinal microbiota and immune system, Nature, 13 Sept .2012, Volume489
  11. Nathan C, Ding A, Nonresolving inflammation, Cell. 2010 Mar19;140(6);871-82
  12. Nesterov, V. I, Method of biolocation studies accuracy increasing. Current Topics , Journal of the Neurology, Psychiatry and Related Disciplines, Vol XX, No 3-4, December 2012, The instity
  13. Nesterov, VI, Main tendencies of non-linear technology development, Acta Medica Medianae 2013; 52(2) 41-44.Perez-Fontan M., et al, Enterococcal peritonitis in peritoneal dialysis patients: last name matters, Peritoneal Dialyse International, 20111, pp 513-517Vo31
  14. Perez-Fontan M., et al, Enterococcal peritonitis in peritoneal dialysis patients: last name matters, Peritoneal Dialyse International, 20111, pp 513-517Vo31
  15. Proal, AD, et al. The human microbiome and autoimmunity. Current Opinion Rheumatology 2013, Vol 25 , number 2, March 2013 :234-240
  16. RB, Bacteria in Crohn’s disease mechanisms of inflammation and therapeutic implications’ Clin Gastroenterol., 2003 May /June;41, Supp.1:37-43
  17. Sood. S., Malhotra M., B.K., Kapil & D, Enterrococcal infections and antimicrobial resistance, Indian Journal Med Res 128, August 2008, pp 111-12118.University of Gothenburg. “An altered gut microbiota can predict diabetes.” ScienceDaily. ScienceDaily, 3 June 2013. <>.
  18. University of Gothenburg. “An altered gut microbiota can predict diabetes.” ScienceDaily. ScienceDaily, 3 June 2013. <>.



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