Testosterone Linked to Arterial Stiffness – Role of Inflammation

By August 13, 2012 Anti-Aging

The decline in dehydroepiandrosterone (DHEA), our anti-aging hormone due to ageing may be one reason behind increased cardiovascular disease risk in ageing populations, and this in turn may be mediated through inflammatory processes.

We can expect many changes as we age, everything from declining hormone levels to elevated chronic disease risk; but what is the reason? And perhaps more importantly – is there a connection? A recent study, awaiting publication in the journal Aging Cell, contributed fascinating knowledge to the field by exploring just this question. Specifically, noticing a decline in serum DHEA concentration occurring alongside an increase in cardiovascular disease risk in ageing individuals, the authors investigated the relationship between these two variables and the mechanism through which this relationship may be mediated. (Source; Bioconcept, August 2012)

Danuta Hulajko Comments

Low testosterone in men and women results in low libido, obesity, muscle waste, low bone density, fertility problems,  hair loss and  nervous  system issues  like reduced mood, concentration, sleep, headache and tinnitus. I have seen in my Clinic increasing number of men and women with low testosterone and quite often cardiovascular and other complications. In men low testosterone also causes erectile dysfunction and prostate atrophy.

There is an extensive range of natural nutritional and herbal supplements which can balance DHEA and increase testosterone in men and women.  While our Clinic can assist you in indentifying and addressing the underlying often complex causes of low testosterone there is a lot YOU can do to prevent or minimize its impact on your life.

Maintaining healthy weight, physical activity, good quality of sleep, minimizing exposure to toxic chemicals, pharmaceutical drugs and intake of alcohol has significant impact on addressing low testosterone. We will provide you with a specific easy to follow diet.